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The wt, gpoA, tpxD and ahpD mutants grew significantly more in nox2 -KO, which also present low ROS levels [22], than in the wt PLB-985 cells (Fig

Further research confirms that ACSL4 suppression in early ischemic stages is time-dependent and likely driven by increased HIF-1, which binds to the ACSL4 promoter and suppresses its expression (137)